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Home >> Homeopathic Treatment For Cirrhosis of Liver

Homeopathic Treatment For Cirrhosis of Liver

Definition: The term cirrhosis is applied to chronic diffuse liver disease of varied etiology, and characterized by hepatic cell necrosis, proliferation of connective tissue and nodular regene- ration or in other words abnormal reconstruction of lobular architecture and disturbed hepatic circulation.
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Etiological Classification :

  • Cryptogenic-In many patients the factors that lead to the development of cirrhosis are unknown.
  • Following infections-(a) Viral hepatitis B. (b) chronic active hepatitis. (c) Congenital syphilis.
  • Induced by alcohol and drugs-(a) Alcohol-produces liver damage if consumed in large amounts. Some pro-gress to cirrhosis. (b) Drugs-Methotraxate. At times axyphenisatin, monoamine oxidase inhibitors, halothane.
  • Following biliary obstruction-Stones, stricture, tumours, biliary atresia.
  • Passive congestion-Chronic congestive cardiac failure, chronic venous outflow obstruction, e.g., Budd-Chiari syn- drome, veno-acclusive disease, sickle-cell anaemia.
  • Malnutrition-(a) Protein deficiency-Probably liver affected by malnutrition is more sensitive to injury which leads to cirrhosis. (b) Food contaminants-Sustained intake of plant and fungal alkaloids may lead to cirrhosis possibly by interfering with protein synthesis in the liver. A possible example is aflatoxin found in nuts and vegetables contaminated with Aspergillus flavus flavus mycotoxins which may explain the high incidence of cirrhosis of unestablish-ed etiology in tropical areas.
  • Congenital-(i) Hereditary haemorrhagic telangiectasia. (ii) Inborn errors of metabolism-(a) Galactosuria. (b) Type IV glycogen storage disease. (c) Tyrosinosis. (d) Alpha-antitrypsin deficiency. (e) Thallassemia. (f) Wilson's disease. (g) Haemochromatosis. (h) Cystic fibrosis.
  • Miscellaneous-(a) Primary biliary cirrhosis. (b) Indian childhood cirrhosis. (c) Sarcoid cirrhosis.

Clinical features :

Symptoms and signs-of cirrhosis depend mainly on-(a) Liver cell insufficiency and (b) Portal hypertension.
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  • Onset(a) Vague with anorexia, dyspepsia, weight loss, malaise and loss of libido. (b) Dramatic with jaundice, ascites or haematemesis. (c) Asymptomatic with hepato-megaly. (d) Miscellaneous-Swelling of ankles, diarrhea, low grade fever.
  • Hepatic-(a) Hepatomegaly-Liver may be palpable, non-tender, shrinks as disease advances. (b) Jaundice- Uncom-mon, when present it may be due to hepatocellular failure or intrahepatic cholestasis. (c) Ascites-develops gradu-ally, rarely suddenly following trauma to abdomen, gut bleeding, acute infection or portal thrombosis. Mechanism.
  • Digestive-(a) Haematemesis-due to rupture of oeso-phageal varices may be the presenting symptom. Other evidence of collateral circulation are dilated periumbilical veins and haemorrhoids. (b) Peptic ulcer. (c) Parotid enlargement. (d) Pancreatitis.
  • Endocrine-(i) In male-Gynecomastia, testicular atrophy, femininisation, reduced body hair, impotence. (b) In female-Lowered libido and asually atrophy of the breasts.
  • Haematological-(a) Purpura. (b) Anaemia-may be caused by bleeding, impaired conversion of folic acid into folinic acid, impaired metabolism of vitamin B12 and haemolysis.
  • Dermatological and musculoskeletal - Arterial spiders - central arteriole from which radiate numerous small vessels. May be seen in necklace area, face, forearm and dorsum of hand. May disappear with improving hepatic function.
  • Circulatory-(a) Hyperdynamic circulation due to increased blood volume, associated anaemia, A-V shunting within the lungs and excwssive vasodilator material due to failure of detoxification by the damaged liver. (b) Clubbing. (c) Cyanosis.
  • Neurological-(a) Portosystemic ancephalopathy. (b) Peripheral neuropathy.
  • Renal-Hepato-renal syndrome. About 80% of patients dying from cirrhosis have renal failure and in majority of thses it is functional renal failure / acute tubular necrosis or diuretic-induced.
  • 10. Miscellaneous-(a) Fever-low grade common due to bacteremia, or continuing hepatic cell necrosis, or infected ascites, or rarely due to development of hepatoma. (b) Hydrothorax-may occur in right pleural cavity.

Investigations :

  • Blood-(a) Anaemia normocytic, normochromic; may be hypochromic if gastric haemorrhage. Occasionally macro-cytic. (b) Low white cell count or reduced platelets due to hypersplenism. (c) Raised ESR from abnormal serum proteins. (d) Mitochondrial antibodies in biliary cirrhosis.
  • Liver function tests- may be normal in patients with compensated cirrhosis. Usually slight increase in serum trans- aminase, alkaline phosphatase and gamma globulin levels and fall in serum albumin levels and fall in serum albumin level.
  • Radiology
    • (a) Barium swallow
    • (b) Transplenic portal venography
    • (c) Selective celiac arterio-portography
    • (d) Umbilical venography
    • (e) Liver scan
  • Histological-Liver biopsy shows typical changes.
  • Mechanical
    • (a) Proctoscopy
    • (b) Laparoscopy
    • (c) Estimation of portal pressure
    • (d) Electroencephalogram
  • Complications
    • Due to portal hypertension
    • Due to liver cell dysfunction
    • Due to formation of regeneration nodules
    • Mechanical due to ascites
    • Due to infection
    • Hypersplenism
    • Chronic renal failure
  • Differential Diagnosis :
    • Depending upon clinical presentation of
    • Hepatomegaly
    • Haematemesis
    • Splenomegaly
    • Jaunice
    • Ascites
  • Management : is palliative
    • Rest in bed-till improvement continues
    • Diet-Low salt. Total daily intake of 2000 calories with protein intake of 120 gm. If patient can tolerate it. Fats and carbohydrates in normal amounts. Vitamin B complex.
    • Treatment of liver disease-Abstinence from alcohol in the alcoholic. Steroids in case of chronic active hepatitis. Appropriate therapy in case of Wilson's disease and haemochromatosis.
    • Drugs-(a) Corticosteroids- may help patient with active posthepatitis cirrhosis. Prednisolone is continued in a small maintenance dose of 10 mg. daily for many months. (b) Immunosuppressive agents-in case of troublesome side effects of corticosteroids.
    • Symptomatic treatment
      • (i) Anaemia
      • (ii) Restlessness
      • (iii)Ascites
      • (iv) Haematemesis
      • (i) Anaemia
The changes you will need to make will depend on how well your liver is working. Talk to your doctor about the kind diet that is best for you so that you get the right amount of nutrition.

General recommendations for patients with severe liver disease include:
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  • Eat large amounts of carbohydrate foods. Carbohydrates should be the major source of calories in this diet.
  • Eat a moderate intake of fat, as prescribed by the health care provider. The increased carbohydrates and fat help prevent protein breakdown in the liver.
  • Have about 1 gram of protein per kilogram of body weight. This means that a 154-pound (70-kilogram) man should eat 70 grams of protein per day. This does not include the protein from starchy foods and vegetables. A person with a badly damaged liver may need to eat less protein. Talk to your doctor about your protein needs.
  • Take vitamin supplements, especially B-complex vitamins.
  • Reduce the amount of salt you consume (typically less than 1500 milligrams per day) if you are retaining fluid.

Diet plane for cirrhosis of Liver:

  • Breakfast
    • 1 orange
    • Cooked oatmeal with milk and sugar
    • 1 slice of whole-wheat toast
    • Strawberry jam
    • Coffee or tea
  • Lunch
    • 4 ounces of cooked lean fish, poultry, or meat
    • A starch item (such as potatoes)
    • A cooked vegetable
    • Salad
    • 2 slices of whole-grain bread
    • 1 tablespoon of jelly
    • Fresh fruit
    • Milk
  • Mid-Afternoon Snack
    • Milk with graham crackers
  • Dinner
    • 4 ounces of cooked fish, poultry, or meat
    • Starch item (such as potatoes)
    • A cooked vegetable
    • Salad
    • 2 whole-grain rolls
    • Fresh fruit or dessert
    • 8 ounces of milk
  • Evening snack
    • Glass of milk or piece of fruit
    • Most of the time you do not have to avoid specific foods.
    • Talk to your doctor if you have questions about your diet or symptoms

Homoeopathic Approach to Cirrhosis of Liver Treatment :

The homoeopathic approach to the Treatment for cirrhosis of Liver patients is more individualistic. This means, homoeopathy believes that migraine is a personality disorder and hence the treatment should be determined only on the basis of in-depth study of the patients' personality. This approach helps treating most cases of migraine successfully.

Homeopathic Treatment